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Computational modeling uncovers new molecular mechanisms controlling T helper cell function
Marketing and Communications - Press Releases
2013, May. 02

BLACKSBURG, Va., May 2, 2013 – Researchers at the Center for Modeling Immunity to Enteric Pathogens at Virginia Bioinformatics Institute believe they have identified the mechanism that turns pro-inflammatory Th17 cells into anti-inflammatory T regulatory cells in the gut. This marks the first time that a comprehensive computational and mathematical model of the mechanisms governing Th cell differentiation has been created and key model-derived predictions have been validated experimentally. The results have been published in PLOS Computational Biology.
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Research Highlight

Anil Vullikanti, PhD

Anil Vullikanti, PhD

The recent recipient of an NSF Early Career Award, Dr. Vullikanti is a Senior Research Associate in the Network Dynamics and Simulation Science Laboratory at VBI. His current interests are at the interface of computer science and modeling and simulation of social and infrastructure systems of social and infrastructure systems, epidemiology, and mobile computing.

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Tue, May 28, 8:00am - Sat, Aug 03, 5:00pm
REU Site: Modeling and Simulation in Systems Biology (MSSB)
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REU Site: Microbiology in the Post Genome Era
Mon, Jul 08, 8:00am - Fri, Jul 12, 5:00pm
High School Summer Internship: Modeling of infectious diseases
Sat, Aug 03, 8:00am - 5:00pm
Symposium: Computationally-driven experimental biology in engineered tissues
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